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Osteoimmunology [electronic resource] / edited by Yongwon Choi.

Por: Tipo de material: TextoTextoSeries Advances in Experimental Medicine and Biology ; 602 | Advances in Experimental Medicine and Biology ; 602Editor: Boston, MA : Springer US, 2007Descripción: XVI, 156 p. online resourceTipo de contenido:
  • text
Tipo de medio:
  • computer
Tipo de soporte:
  • online resource
ISBN:
  • 9780387720098
Trabajos contenidos:
  • SpringerLink (Online service)
Tema(s): Formatos físicos adicionales: Sin títuloClasificación CDD:
  • 616.079 23
Clasificación LoC:
  • QR180-189.5
Recursos en línea:
Contenidos:
Springer eBooksResumen: A Key Regulator of Postnatal Skeletal Remodeling.- Ectodomain Shedding of Receptor Activator of NF-KB Ligand.- The Negative Role Of Ids In Osteoclastogenesis.- Functional Genetic and Genomic Analysis of Modeled Arthritis.- Dexamethsone Suppresses Bone Formation via the Osteoclast.- Immunologic Regulation Of Bone Development.- Pth Regulates The Hematopoietic Stem Cell Niche In Bone.- Regulation Of Hematopoietic Stem Cells In The Osteoblastic Niche.- The Chemokine Cxcl12 And Regulation Of Hsc And Lymphocyte Development In The Bone Marrow Niche.- Osteoclast Precursor Cells.- Interaction with estrogen receptors as treatment of arthritis and osteoporosis.- Novel Signaling Pathways And Therapeutic Targets In Osteoclasts.- The Enigmatic Function of TREM-2 in Osteoclastogenesis.- Role of cell-matrix interactions in osteoclast differentiation.- Positive and negative roles of IL-6, STAT3 and SOCS3 in inflammatory arthritis.- Control of Osteoclast activity and bone loss by IKK subunits: new targets for therapy.- Targeting Osteoporosis And Rheumatoid Arthritis By Active Vaccination Against Rankl.- RANKL Inhibition: From Mice to Men (and Women).
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Schnurri-3: A Key Regulator of Postnatal Skeletal Remodeling -- Ectodomain Shedding of Receptor Activator of NF-?B Ligand -- The Negative Role of IDS in Osteoclastogenesis -- Functional Genetic and Genomic Analysis of Modeled Arthritis -- Dexamethsone Suppresses Bone Formation via the Osteoclast -- Immunologic Regulation of Bone Development -- PTH Regulates the Hematopoietic Stem Cell Niche in Bone -- Regulation of Hematopoietic Stem Cells in the Osteoblastic Niche -- The Chemokine CXCL12 and Regulation of Hsc and B Lymphocyte Development in the Bone Marrow Niche -- Osteoclast Precursor Cells -- Interaction with Estrogen Receptors as Treatment of Arthritis and Osteoporosis -- Novel Signaling Pathways and Therapeutic Targets in Osteoclasts -- The Enigmatic Function of TREM-2 in Osteoclastogenesis -- Role of Cell-matrix Interactions in Osteoclast Differentiation -- Positive and Negative Roles of IL-6, STAT3, and SOCS3 in Inflammatory Arthritis -- Control of Osteoclast Activity and Bone Loss by IKK Subunits: New Targets for Therapy -- Targeting Osteoporosis and Rheumatoid Arthritis by Active Vaccination Against RANKL -- RANKL Inhibition: From Mice to Men (and Women).

A Key Regulator of Postnatal Skeletal Remodeling.- Ectodomain Shedding of Receptor Activator of NF-KB Ligand.- The Negative Role Of Ids In Osteoclastogenesis.- Functional Genetic and Genomic Analysis of Modeled Arthritis.- Dexamethsone Suppresses Bone Formation via the Osteoclast.- Immunologic Regulation Of Bone Development.- Pth Regulates The Hematopoietic Stem Cell Niche In Bone.- Regulation Of Hematopoietic Stem Cells In The Osteoblastic Niche.- The Chemokine Cxcl12 And Regulation Of Hsc And Lymphocyte Development In The Bone Marrow Niche.- Osteoclast Precursor Cells.- Interaction with estrogen receptors as treatment of arthritis and osteoporosis.- Novel Signaling Pathways And Therapeutic Targets In Osteoclasts.- The Enigmatic Function of TREM-2 in Osteoclastogenesis.- Role of cell-matrix interactions in osteoclast differentiation.- Positive and negative roles of IL-6, STAT3 and SOCS3 in inflammatory arthritis.- Control of Osteoclast activity and bone loss by IKK subunits: new targets for therapy.- Targeting Osteoporosis And Rheumatoid Arthritis By Active Vaccination Against Rankl.- RANKL Inhibition: From Mice to Men (and Women).

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